Trauma Related Brain Damage Glutamate and Neurotoxicity
youtu.be
The content segments discuss the role of glutamate and neurotoxicity in trauma-related brain damage. Glutamate is a crucial neurotransmitter in the brain that is necessary for learning and synaptic plasticity. However, excessive glutamate can lead to inflammation and excitotoxicity, damaging nerve cells and contributing to anxiety. Too little glutamate can result in difficulty concentrating and symptoms similar to depression. The balance of glutamate is essential for proper brain functioning.
Chronic stress and trauma can dysregulate the HPA axis, leading to pathological changes in the body and brain. Hypervigilance, a normal reaction to trauma, can result in continuously elevated levels of cortisol and glutamate, which can be toxic to cells. This dysregulation can contribute to symptoms of depression and anxiety.
Excessive glutamate and HPA axis dysregulation are considered core features of stress-related mental and physical illnesses. Systemic inflammation, related to mental health symptoms and autoimmune diseases, can be triggered by high levels of cortisol and glutamate.
The content also discusses the potential use of glutamate antagonists and various nutrients and substances to normalize glutamate levels. Therapeutic approaches, such as EMDR, CPT, and ACT, can help manage intrusive thoughts and create a sense of safety. Mindfulness meditation, exercise, and spending time in nature can also promote brain health and help individuals cope with trauma.
Terpenes, found in plants and essential oils, have neuroprotective effects and can help normalize glutamate levels. However, it is important to use essential oils cautiously and under medical supervision.
Overall, the content emphasizes the importance of managing stress levels, addressing trauma triggers, and promoting a sense of safety to support the restoration of normal glutamate neurotransmission and healthier brain functioning.
The content segments discuss the role of glutamate and neurotoxicity in trauma-related brain damage.